- Genetic Addiction Risk Score (GARS): molecular neurogenetic evidence for predisposition to Reward Deficiency Syndrome (RDS). – PubMed – NCBI
- Neurobiology of KB220Z-Glutaminergic-Dopaminergic Optimization Complex [GDOC] as a Liquid Nano: Clinical Activation of Brain in a Highly Functional C… – PubMed – NCBI
- Hypothesizing That Neuropharmacological and Neuroimaging Studies of Glutaminergic-Dopaminergic Optimization Complex (KB220Z) Are Associated With “Dopamine Homeostasis” in Reward Deficiency Syndrome (RDS)
- The Benefits of Customized DNA Directed Nutrition To Balance The Brain Reward Circuitry And Reduce Addictive Behaviors – PubMed – NCBI
- Neurophysiological Measures and Alcohol Use Disorder (AUD): Hypothesizing Links between Clinical Severity Index and Molecular Neurobiological Pattern – PubMed – NCBI
- Pro-Dopamine Regulator – (KB220) to Balance Brain Reward Circuitry in Reward Deficiency Syndrome (RDS). – PubMed – NCBI
- Genetic addiction risk score (GARS) ™, a predictor of vulnerability to opioid dependence. – PubMed – NCBI
- Global Opioid Epidemic: Doomed To Fail Without Genetically Based Precision Addiction Medicine (Pam™): Lessons Learned From America. – PubMed – NCBI
- Coupling Genetic Addiction Risk Score (GARS) and Pro Dopamine Regulation (KB220) to Combat Substance Use Disorder (SUD). – PubMed – NCBI
Earlier KB220Z Research
- Reward Deficiency Syndrome Studies of KB220 Variants and Molecular Neurobiology
- Enhanced functional connectivity and volume between cognitive and reward centers of naïve rodent brain produced by pro-dopaminergic agent KB220Z.
- rsfMRI effects of KB220Z™ on neural pathways in reward circuitry of abstinent genotyped heroin addicts. – PubMed – NCBI
- Neurogenetics and gene therapy for reward deficiency syndrome: are we going to the Promised Land? – PubMed – NCBI
- Can Genetic Testing Provide Information to Develop Customized Nutrigenomic Solutions for Reward Deficiency Syndrome? – PubMed – NCBI
- Dopamine D2 gene expression interacts with environmental enrichment to impact lifespan and behavior. – PubMed – NCBI
- The Psychoactive Designer Drug and Bath Salt Constituent MDPV Causes Widespread Disruption of Brain Functional Connectivity. – PubMed – NCBI
- Common Neurogenetic Diagnosis and Meso-Limbic Manipulation of Hypodopaminergic Function in Reward Deficiency Syndrome (RDS): Changing the Recovery La… – PubMed – NCBI
- Neuronutrient Amino-Acid Therapy Protects Against Reward Deficiency Syndrome: Dopaminergic Key to Homeostasis and Neuroplasticity. – PubMed – NCBI
- Low-Resolution Electromagnetic Tomography (LORETA) of changed Brain Function Provoked by Pro-Dopamine Regulator (KB220z) in one Adult ADHD case. – PubMed – NCBI
- Dopamine homeostasis: brain functional connectivity in reward deficiency syndrome. – PubMed – NCBI
- KB220Z™ a Pro-Dopamine Regulator Associated with the Protracted, Alleviation of Terrifying Lucid Dreams. Can We Infer Neuroplasticity-induced Changes… – PubMed – NCBI
- Lyme and Dopaminergic Function: Hypothesizing Reduced Reward Deficiency Symptomatology by Regulating Dopamine Transmission. – PubMed – NCBI
- Overcoming qEEG abnormalities and reward gene deficits during protracted abstinence in male psychostimulant and polydrug abusers utilizing putative d… – PubMed – NCBI
- A Shared Molecular and Genetic Basis for Food and Drug Addiction: Overcoming Hypodopaminergic Trait/State by Incorporating Dopamine Agonistic Therapy… – PubMed – NCBI
- Clinically Combating Reward Deficiency Syndrome (RDS) with Dopamine Agonist Therapy as a Paradigm Shift: Dopamine for Dinner? – PubMed – NCBI
- Pro-Dopamine Regulator – (KB220Z) to Balance Brain Reward Circuitry in Reward Deficiency Syndrome (RDS). – PubMed – NCBI
- The Food and Drug Addiction Epidemic: Targeting Dopamine Homeostasis. – PubMed – NCBI
- Click here for Clinical Studies from 1973-2016